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Article Addendum
Role of HEMIVENATA and the Ubiquitin Pathway in Venation Pattern Formation
Hector Candela, María Magdalena Alonso-Peral, María Rosa Ponce and José Luis Micol
volume 2 | issue 4
july/august 2007Pages: 258 - 259
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Elegant work by others has highlighted the importance of auxin transport in venation patterning, an idea substantiated by the severe effects of auxin polar transport inhibitors and by the mutant phenotype and expression patterns associated with the auxin efflux transporter PIN-FORMED1 (PIN1). It is striking, therefore, that little attention has been paid to the venation patterns of mutants insensitive to this hormone, since both auxin transport and perception are crucial components in theoretical models of vascular patterning. Our finding that HEMIVENATA (HVE) is the same gene as CAND1 confirms the role of ubiquitin-mediated auxin perception in vascular patterning and sets the stage for a re-examination of the leaf venation phenotypes of other auxin-resistant mutants and additional components of the ubiquitin pathway.
Authors
Hector Candela
UC-Berkeley
María Magdalena Alonso-Peral
Universidad Miguel Hernández, Alicante, Spain
María Rosa Ponce
Universidad Miguel Hernández, Alicante, Spain
José Luis Micol
Universidad Miguel Hernández, Alicante, Spain
We now provide open access to journal articles published online for one year or more. This article may be downloaded at the following link:
Download PDF If the document does not open, please right-click on the link (control-click on a Macintosh) and select the option to save the file to disk.