Editorials: Cell Cycle Features
Combination of isoform-selective histone/protein deacetylase inhibitors improves Foxp3+ T-regulatory cell function
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Volume 11, Issue 18 September 15, 2012
Pages 3351 - 3352
http://dx.doi.org/10.4161/cc.21876
Keywords: HDAC6, HDAC9, Sirt1, immunotherapy, transplantation
Authors: Ulf H. Beier, Liqing Wang and Wayne W. Hancock
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- Ulf H. Beier
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Division of Nephrology; Department of Pediatrics; Children’s Hospital of Philadelphia; Philadelphia, PA USA; University of Pennsylvania School of Medicine; Philadelphia, PA USA
- Liqing Wang
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Division of Transplant Immunology; Department of Pathology and Laboratory Medicine and Biesecker Center for Pediatric Liver Disease; Children’s Hospital of Philadelphia; Philadelphia, PA USA
- Wayne W. Hancock
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Corresponding author: whancock@mail.med.upenn.edu
University of Pennsylvania School of Medicine; Philadelphia, PA USA; Division of Transplant Immunology; Department of Pathology and Laboratory Medicine and Biesecker Center for Pediatric Liver Disease; Children’s Hospital of Philadelphia; Philadelphia, PA USA
Abstract:
Comment on: Beier UH, et al. Sci Signal 2012; 5:ra45.
Editorials: Cell Cycle Features to:
UH Beier, L Wang, R Han, T Akimova, Y Liu, WW Hancock. Histone deacetylases 6 and 9 and sirtuin-1 control Foxp3+ regulatory T cell function through shared and isoform-specific mechanisms. Sci Signal 2012; 5: ra45-ra45
PMID: 22715468 DOI: 10.1126/scisignal.2002873
Received: July 17, 2012; Accepted: July 18, 2012; Published Online: August 23, 2012
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