• Cell cycle synchronization of leukemia inhibitory factor (LIF)-dependent porcine-induced pluripotent stem cells and the generation of cloned embryos
  • Dissecting the first and the second meiotic divisions using a marker-less drug-hypersensitive fission yeast
  • CAPER, a novel regulator of human breast cancer progression
  • Multi-gene fluorescence in situ hybridization to detect cell cycle gene copy number aberrations in young breast cancer patients

Cell cycle synchronization of leukemia inhibitory factor (LIF)-dependent porcine-induced pluripotent stem cells and the generation of cloned embryos

Ye Yuan, Kiho Lee, Kwang-Wook Park, Lee D Spate, Randall S Prather, Kevin D Wells and R Michael Roberts

Dissecting the first and the second meiotic divisions using a marker-less drug-hypersensitive fission yeast

Yuki Aoi, Masamitsu Sato, Takashi Sutani, Katsuhiko Shirahige, Tarun M Kapoor and Shigehiro A Kawashima

CAPER, a novel regulator of human breast cancer progression

Isabelle Mercier, Donna M Gonzales, Kevin Quann, Timothy G Pestell, Alexander Molchansky, Federica Sotgia, James Hulit, Ricardo Gandara, Chenguang Wang, Richard G Pestell, Michael P Lisanti and Jean-François Jasmin

Multi-gene fluorescence in situ hybridization to detect cell cycle gene copy number aberrations in young breast cancer patients

Chunyan Li, Jingchao Bai, Xiaomeng Hao, Sheng Zhang, Yunhui Hu, Xiaobei Zhang, Weiping Yuan, Linping Hu, Tao Cheng, Anders Zetterberg, Mong-Hong Lee and J Zhang

Current Issue

Cell Cycle

April 15, 2014

Volume 13, Issue 8

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About the cover image
Immunofluorescence results demonstrated that endogenous E2F1 were partially colocalized with endogenous RARα in U2OS cells, suggesting E2F1 directly interacts with RARα. We further found that E2F1 promotes ubiquitination-mediated degradation of RARα and inhibits RARα-mediated osteogenic differentiation. For more information, see Zhang et al., pp. 1218–9.

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