Commentary
The mismatch repair and base excision repair pathways: An opportunity for individualized (personalized) sensitization of cancer therapy
Downloads and Tools
Volume 8, Issue 12 June 15, 2009
Pages 1164 - 1166
http://dx.doi.org/10.4161/cbt.8.12.8750
Keywords: base excision repair pathway, iododeoxyuridine, methyl binding domain protein 4 (MBD4/MED1), mismatch repair pathway, thymine DNA glycosylase, uracil DNA glycosylase
Authors: Sean P. Collins and Anatoly Dritschilo
View affiliations Hide affiliations
- Sean P. Collins
-
Departments of Radiation Medicine and Oncology; Lombardi Comprehensive Cancer Center; Georgetown University Medical Center; Washington, DC USA
- Anatoly Dritschilo
-
Corresponding author: dritscha@georgetown.edu
Departments of Radiation Medicine and Oncology; Lombardi Comprehensive Cancer Center; Georgetown University Medical Center; Washington, DC USA
Commentary to:
MA Aziz, JE Schupp, TJ Kinsella. Modulation of the activity of methyl binding domain protein 4 (MBD4/MED1) while processing iododeoxyuridine generated DNA mispairs. Cancer Biol Ther 2009; 8: 1156-63
PMID: 19395862 DOI: 10.4161/cbt.8.12.8536
Preview: